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This is your brain on Facebook. Researchers at the Stanford University School of Medicine used concepts borrowed from the popular social networking site to analyze the brains of people with Alzheimer’s disease. They found that patients’ brains were less well-connected than the brains of people without the disorder.

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Research in fruit flies has shown that enhancing the production of a protein called neprilysin can reduce the formation of plaques and neuron death associated with Alzheimer’s, at the expense of reducing the flies’ lifespan. The buildup of amyloid-beta protein plaques within the brain is a major hallmark of Alzheimer’s, and one that is believed to contribute to disease progression.

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A recent study found that about 10 percent of apparently healthy middle-aged participants with no symptoms of stroke were injured from “silent strokes,” researchers report in Stroke: Journal of the American Heart Association. Silent cerebral infarction (SCI), or “silent stroke,” is a brain injury likely caused by a blood clot interrupting blood flow in the brain.

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Researchers have discovered the second, strong genetic risk factor for developing late-onset Alzheimer’s disease, according to a new report in the June 27th issue of the journal Cell, a Cell Press publication.The newly discovered gene, which previously had no known function, is predominantly active in a region of the brain that is hit early in the disease, where it acts as a channel for calcium, they show.

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Anavex Life Sciences Corp. (”ANAVEX”) (OTCBB: AVXL) announces its participation at the International Conference on Alzheimer’s Disease 2008 (July 26-31, 2008, Chicago, USA), where it will present results obtained with ANAVEX 1-41 demonstrating a protective effect against the neurotoxicity of amyloid (beta)25-35 peptide (A(beta)25-35) in mice.

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A new study uncovers a mechanism that directly links mutations that cause early onset Alzheimer’s disease (AD) with aberrant calcium signaling. The research, published by Cell Press in the June 26th issue of the journal Neuron, provides exciting molecular insights into the pathology of AD and may lead to new treatment strategies.AD is a devastating neurodegenerative disease that affects early 18 million people in the world.

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Using new scientific techniques, scientists have unlocked the cascade of molecular events that lead to Alzheimer’s disease. The scientific findings published in the latest edition of Nature Medicine suggest a potential new target for the development of drug therapies to fight the irreversible and degenerative disease which affects some 29.8 million people worldwide. The total worldwide societal cost of dementia was estimated at somewhere in the region of US$315.4 billion in 2005.

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Scottish researchers funded by the UK’s leading dementia research charity, the Alzheimer’s Research Trust, have found a link between childhood IQ and vascular dementia later in life.Their research, published in the journal Neurology, found that lower childhood intelligence increases the risk of vascular dementia, a disease which currently affects 112,000 people in the UK.

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Thousands of people with dementia are being forced to pay large amounts for vital care, which is often poor quality, according to a new report published by Alzheimer’s Society. The Dementia Tax shows that over two thirds of people with dementia are paying for basic care such as help with washing, dressing and going to the toilet that they require as a result of their medical condition. The majority of people are paying at least £100 a week towards the costs of care.

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Children with lower IQs are more likely decades later to develop vascular dementia than children with high IQs, according to research published in the June 25, 2008, online issue of Neurology®, the medical journal of the American Academy of Neurology.The most common type of dementia after Alzheimer’s disease, vascular dementia occurs when blood flow to the brain is impaired.

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